Browsing by Author "Prasai, Parikshit"
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Publication Autoimmune Hemolytic Anemia with Autoimmune Hypothyroidism: A Case Report(Nepal Medical Association, 2023) Karki, Prakriti; Prasai, Parikshit; Chetri, Vivek Acharya; Gautam, Arun; Maskey, RobinAbstract Autoimmune hemolytic anaemia is a relatively rare disorder caused by autoantibodies directed against self-red blood cells. Though autoimmune thyroid disease is associated with other autoimmune diseases, only a few cases of Hashimoto's thyroiditis with autoimmune hemolytic anaemia have been reported. We present a case of a 22-year-old woman, a known case of Hashimoto’s thyroiditis whose serum demonstrated antibodies against red blood cells. Blood investigations were done which showed findings suggestive of hemolytic anemia. She was managed with blood transfusion, thyroxine and steroids. Our study may guide physicians toward possible hemolytic anaemia while treating Hashimoto thyroiditis.Publication Cecal Perforation Following Intraperitoneal Abscess after Anti-tubercular Therapy: A Case Report(Nepal Medical Association, 2023) Prasai, Parikshit; Joshi, Anjali; Poudel, Santosh; K.C., Sarjan; Pahari, RabinAbstract Abdominal tuberculosis is defined as infection of gastrointestinal tract, peritoneum, abdominal solid organs, and/or abdominal lymphatics constituting approximately 12% of extra-pulmonary tuberculosis cases. Intestinal perforation is an acute presentation of abdominal tuberculosis. Intestinal perforation can occur before or at the beginning of anti-tubercular therapy. It is considered to be a paradoxical reaction if it occurs during or after treatment. Intestinal perforation is uncommon but serious and life-threatening as complication-mortality rate secondary to perforation are estimated to be >30%. We present a case of an 18-year-old female who developed cecal perforation following an intraperitoneal abscess after completion of anti-tubercular therapy for intestinal tuberculosis. She was a known case of intestinal tuberculosis. She had undergone pigtail catheterisation for an intraperitoneal abscess and completed 18 months of anti-tubercular therapy after which she developed cecal perforation. A paradoxical response was observed following the completion of anti-tubercular therapy. Early diagnosis and treatment reduce the complications and mortality rates of cecal perforation due to abdominal tuberculosis.